GRP78 as a Target for Prevention of Age-related Cataract By Regulation of Reticulum Endoplasmic Stress

Please use this identifier to cite or link to this item: https://repository.unej.ac.id/xmlui/handle/123456789/112324

Full metadata record

DC Field	Value	Language
dc.contributor.author	CAHYANA, Nugraha Wahyu	-
dc.date.accessioned	2023-02-22T07:26:06Z	-
dc.date.available	2023-02-22T07:26:06Z	-
dc.date.issued	2023-01-01	-
dc.identifier.uri	https://repository.unej.ac.id/xmlui/handle/123456789/112324	-
dc.description.abstract	Cataract is the most common causes of blindness worldwide. The most common kind of cataract is age-related cataract. Measures to prevent the development of cataracts are urgently needed. Significant data suggests that increased endoplasmic reticulum stress is a significant role in cataract development. The goal of this review is to investigate the involvement of endoplasmic reticulum as a factor in cataracts caused by the buildup of unfolded proteins from lens epithelial cells. GRP78 is one of the key signs of endoplasmic reticulum stress, and it promotes the UPR to limit the increase in unfolded protein levels. The significance of GRP78 signaling in cataract prevention is discussed in this paper.	en_US
dc.language.iso	en	en_US
dc.publisher	International Journal of Innovative Research in Multidisciplinary Education	en_US
dc.subject	Cataracts	en_US
dc.subject	reticulum endoplasmic stress	en_US
dc.subject	GRP78	en_US
dc.subject	UPR	en_US
dc.title	GRP78 as a Target for Prevention of Age-related Cataract By Regulation of Reticulum Endoplasmic Stress	en_US
dc.type	Article	en_US
Appears in Collections:	LSP-Jurnal Ilmiah Dosen

Files in This Item:

File	Description	Size	Format
KEDOKTERAN_JURNAL_GRP78 as a Target for Prevention of Age-related Cataract By Regulation of Reticulum Endoplasmic Stress.pdf		655.59 kB	Adobe PDF	View/Open

DSpace JSPUI